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Glioblastoma multiforme is a diffusely infiltrating primary neoplasm of glial origin. Its uniformly fatal prognosis has not changed appreciably over the past decade, and the only modifiable factor associated with survival is the extent of surgical resection. In the absence of a concomitant lesion that is likely to change local treatment, the factors that predict disease recurrence include extensive tumor involvement of white matter tracts and the pattern of invasion of the neuropil. The molecular pathogenesis of glioblastoma is uncertain, but recent advances in understanding the genetic alterations in this tumor and the relationships between these molecular changes and the biologic behavior of the tumor provide a basis for designing therapeutic strategies that will improve long-term survival.Regulation of growth-related signaling events in PC12 cells by the rat homolog of the mouse insulin-like growth factor binding protein, IGFBP-3.
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